Metabolic response to acceleration in man.

LIPMAN, R. L., F. ULVEDAL, W. K. BROWN, S. D. LEVERETT, F. R. LECOCQ and J. J. SCHNURE. Metabolic response to acceleration in man. Aerospace Med. 41 (3) :905-90B. 1970. The glucose analogue 2-deoxy-I)-glucose {2-1X5) was used as a means of producing intracellular bypoglycemia in eight normal male volunteer subjects during a control period, immediately following their initial exposure to acceleration (acute acceleration) and following a fourth acceleration exposure (chronic accelerv tion). Plasma glucose, free fatty acids, serum tmmunoreactive growth hormone, plasma cortisol and urinary epincphrine and norepinephrine were measured prior to and following the infusions of 2-1X3. There were significant depressions in glucose, urinary epinephrine, cortisol and free fatty acids following acceleration thai ver* not seen when compared to basal, un-provoked levels. Even with the modest acceleration stresses used in this study discernible changes in gluco-regulatory hormone reserves were unrovered.